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from The Truth About Cancer

Is Nitric Oxide a Cancer-Healing Powerhouse?

Nitric Oxide is a very powerful genetic regulator but can it suppress cancer tumor growth? The most current research is positive! Read all about it by clicking on the image or pin it for later.


In 1998, three scientists were awarded the Nobel Prize in Physiology of Medicine for their work on Nitric Oxide. Now called “The Molecule of Life,” the Nitric Oxide in Kyanis Nitro FX and Nitro Extreme has been shown to positively influence: Blood Circulation and Heart Health, Inflammation and Joints, Brain and Cognitive Function, Digestion and nutrient absorption, and Sexual Function and Libedo.


This image of the Mars night side shows ultraviolet emission from nitric oxide (abbreviated NO). The emission is shown in false color with black as low values, green as medium, and white as high. These emissions track the recombination of atomic nitrogen and oxygen produced on the dayside, and reveal the circulation patterns of the atmosphere.


Role of Polymorphisms of Inducible Nitric Oxide Synthase and Endothelial Nitric Oxide Synthase in Idiopathic Environmental Intolerances


August 2016: The association of endothelial nitric oxide synthase (eNOS) G894T gene polymorphism with responsiveness to a selective α1-blocker in men with benign prostatic hyperplasia related lower urinary tract symptoms

Steps involved in the synthesis of nitric oxide ([NO] see text for details). cGMP = cyclic guanosine monophosphate; NMDA = N-methyl-D-aspartic acid; nNOS = nitric oxide synthase isoform I.


Try this site for more information on NOS supplement. Nitric Oxide is a gas formed in the body by the breakdown of L-Arginine to L-Citrulline through an enzyme group called Nitric Oxide Synthase (NOS). Although NO2 is used for many purposes, its ability to increase blood flow is seen. Therefore opt for the best NOS supplement and avail the benefits. Follow Us :

from Healthinnovations

New treatment strategy for muscular dystrophy sound in preclinical phase.

Pictured are isolated adult mouse cardiomyocytes labeled for dystrophin (red), neuronal nitric oxide synthase (nNOS) (green), and nuclei (blue). Unlike skeletal muscle, where nNOS physically binds to the dystrophin, nNOS does not directly bind to dystrophin. University of Michigan researchers identified the role of the AMPK pathway for increasing nitric oxide production in mechanically stretched muscle cells.


Macrophages and neutrophils express many surface receptors that may bind microbes for subsequent phagocytosis; select examples of such receptors are shown. Microbes are ingested into phagosomes, which fuse with lysosomes, and the microbes are killed by enzymes and several toxic substances produced in the phagolysosomes. The same substances may be released from the phagocytes and may kill extracellular microbes (not shown). iNOS , Inducible nitric oxide synthase; NO , nitric oxide; ROS…