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DRVVT: used to screen for LAC, because it's a phosophlipid-dependent reaction involving the conversion of X-->Xa.  If you have a LAC, all your PL is bound by antibody and you will not form a clot.  Should correct with mixing. Use platelet poor plasma always in coag reactions.

DRVVT: used to screen for LAC, because it's a phosophlipid-dependent reaction involving the conversion of X-->Xa. If you have a LAC, all your PL is bound by antibody and you will not form a clot. Should correct with mixing. Use platelet poor plasma always in coag reactions.

Natural cures for emphysema (COPD) http://www.omegaxl.com/blog/copd-omega-xl-helps/?GHW_affid=MLIFE

Natural cures for emphysema (COPD) http://www.omegaxl.com/blog/copd-omega-xl-helps/?GHW_affid=MLIFE

Lymphopenia can be caused by SLE, HIV, SARS, Rituxan, steroids and congenital immunodeficiency (Brutons, SCID, diGeorge).

Lymphopenia can be caused by SLE, HIV, SARS, Rituxan, steroids and congenital immunodeficiency (Brutons, SCID, diGeorge).

this slide is just to say that Protein C and S deficiency are more rare, and are usually acquired in liver disease, vit K defiency, warfarin, DIC, sepsis, etc. AT-3 also in same boat.  Protein C testing can be clot based or chromogenic- we will do chromogenic since it has less interference.

this slide is just to say that Protein C and S deficiency are more rare, and are usually acquired in liver disease, vit K defiency, warfarin, DIC, sepsis, etc. AT-3 also in same boat. Protein C testing can be clot based or chromogenic- we will do chromogenic since it has less interference.

Hermansky-Pudlak syndrome is a rare autosomal recessive disorder characterized by oculocutaneous albinism, and lysosomal ceroid deposition.  There is additionally a platelet storage pool defect evident on testing and electron microscopy (absent dense bodies)

Hermansky-Pudlak syndrome is a rare autosomal recessive disorder characterized by oculocutaneous albinism, and lysosomal ceroid deposition. There is additionally a platelet storage pool defect evident on testing and electron microscopy (absent dense bodies)

Figure 2 from Idiopathic Thrombocytopenia with Iron Deficiency Anemia, in Clinical Medicine Insights: Blood Disorders http://la-press.com/article.php?article_id=3634

Figure 2 from Idiopathic Thrombocytopenia with Iron Deficiency Anemia, in Clinical Medicine Insights: Blood Disorders http://la-press.com/article.php?article_id=3634

Schematic diagram of the blood coagulation and protein C pathways. In the blood coagulation pathway, thrombin acts to convert factor XI to XIa, VIII to VIIIa V to Va, fibrinogen to fibrin. In addition, thrombin promotes platelet activation and aggregation via activation of protease-activated receptors on the cell membrane of the platelet. Thrombin also cross over into the protein C pathway by converting protein C into APC. APC in turn converts factor V into Vi, and VIIIa into VIIIi. Finally…

Schematic diagram of the blood coagulation and protein C pathways. In the blood coagulation pathway, thrombin acts to convert factor XI to XIa, VIII to VIIIa V to Va, fibrinogen to fibrin. In addition, thrombin promotes platelet activation and aggregation via activation of protease-activated receptors on the cell membrane of the platelet. Thrombin also cross over into the protein C pathway by converting protein C into APC. APC in turn converts factor V into Vi, and VIIIa into VIIIi. Finally…

Convenient, cheap and comfy!

Convenient, cheap and comfy!

Bleeding Disorder Mini Medical ID. This might be better than getting one that says Von Willebrand's Disease. It's easier than explaining/answering a bunch of questions about vWD.

Bleeding Disorder Mini Medical ID. This might be better than getting one that says Von Willebrand's Disease. It's easier than explaining/answering a bunch of questions about vWD.

PFA 100.  First, do COL:EPI. If abnormal, do COL:ADP.  If normal = ASA.  If abnormal, do more testing.  According to coag meeting, the only reason for PFA 100 is to rapidly identify a more severe platelet disorder like B-S or Glanzmans.

PFA 100. First, do COL:EPI. If abnormal, do COL:ADP. If normal = ASA. If abnormal, do more testing. According to coag meeting, the only reason for PFA 100 is to rapidly identify a more severe platelet disorder like B-S or Glanzmans.

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